Firstly, and this is quite obvious, it is not available to confirmation by direct observation; I have not been seen as behaving cortical blood vessels during migraine aura, and very controversial, that conjunctival vascular changes are adequate to model the behavior of intracranial vessels. Moreover, changes in blood flow in the past is not always observed during migraine aura. Secondly, there is often a coincidence stage aura and headache aura or recurrence of headaches against the background (i.e. the expansion stage vessels). This important fact was not taken into account the Wolf, and it is impossible to reconcile with the theory of the two stages of the migraine attack. Indeed, there may be polisimptomnaya aura, which has both features of excitation and inhibition. The variability of the aura, which can take one or the other form, contrary to the theory postulated in coronary particular vulnerability of the occipital cortex. It is difficult or even impossible to understand, as a "very peculiar function disorders", which is mentioned Gowers - characteristic forms and moving cattle, the vibrational properties of flicker and paresthesias and TD, - can be explained simply by ischemia alone: ??those sensory hallucinations that occur during ischemia in the basilar artery during angiography or vertebral arteries, are very simple and transient, and in addition, do not have specific symptoms of migraine cattle. Of great effect produced by vasoactive drugs sometimes against the duration and intensity of cattle, says nothing about the nature of the process underlying migraine, but only demonstrates the effect of the summation of two neurophysiological disorders. It is known that with a decrease in cerebral perfusion pressure, as, for example, against the background of orthostatic hypotension, syncope occur, dizziness, sometimes "flies flashing before the eyes," and so on. D., But the clinical picture is very different from migraine aura.